[1]Fink SL,Cookson BT.Apoptosis,pyroptosis,and necrosis:Mechanistic description of dead and dying eukaryotic cells[J].Infect Immun,2005,73(4):1907-1916.
[2]Cookson BT,Brennan MA.Pro-inflammatory programmed cell death[J].Trends Microbiol,2001,9(3):113-114.
[3]Shi J,Zhao Y,Wang K,et al.Cleavage of GSDMD by inflammatory caspases determines pyroptotic cell death[J].Nature,2015,526(7575):660-665.
[4]Aglietti RA,Estevez A,Gupta A,et al.GsdmD p30 elicited by caspase-11 during pyroptosis forms pores in membranes[J].Proc Natl Acad Sci USA,2016,113(28):7858-7863.
[5]Ding J,Wang K,Liu W,et al.Pore-forming activity and structural autoinhibition of the gasdermin family[J].Nature,2016,535(7610):111-116.
[6]Shi J,Gao W,Shao F.Pyroptosis:Gasdermin-mediated programmed necrotic cell death[J].Trends Biochem Sci,2017,42(4):245-254.
[7]Galluzzi L,Vitale I,Aaronson SA,et al.Molecular mechanisms of cell death:Recommendations of the nomenclature committee on cell death 2018[J].Cell Death Differ,2018,25(3):486-541.
[8]Wang Y,Gao W,Shi X,et al.Chemotherapy drugs induce pyroptosis through caspase-3 cleavage of a gasdermin[J].Nature,2017,547(7661):99-103.
[9]Mascarenhas DPA,Cerqueira DM,Pereira MSF,et al.Inhibition of caspase-1 or gasdermin-D enable caspase-8 activation in the Naip5/NLRC4/ASC inflammasome[J].PLoS Pathog,2017,13(8):e1006502.
[10]Orning P,Weng D,Starheim K,et al.Pathogen blockade of TAK1 triggers caspase-8-dependent cleavage of gasdermin D and cell death[J].Science,2018,362(6418):1064-1069.
[11]Sarhan J,Liu BC,Muendlein HI,et al.Caspase-8 induces cleavage of gasdermin D to elicit pyroptosis during Yersinia infection[J].Proc Natl Acad Sci USA,2018,115(46):E10888-E10897.
[12]Fujikawa K,Shiraki K,Sugimoto K,et al.Reduced expression of ICE/caspase1 and CPP32/caspase3 in human hepatocellular carcinoma[J].Anticancer Res,2000,20(3B):1927-1932.
[13]Chu Q,Jiang Y,Zhang W,et al.Pyroptosis is involved in the pathogenesis of human hepatocellular carcinoma[J].Oncotarget,2016,7(51):84658-84665.
[14]Yan W,Chang Y,Liang X,et al.High-mobility group box 1 activates caspase-1 and promotes hepatocellular carcinoma invasiveness and metastases[J].Hepatology,2012,55(6):1863-1875.
[15]Higai K,Miyazaki N,Azuma Y,et al.Interleukin-1beta induces sialyl Lewis X on hepatocellular carcinoma HuH-7 cells via enhanced expression of ST3Gal IV and FUT VI gene[J].FEBS Lett,2006,580(26):6069-6075.
[16]Wei Q,Mu K,Li T,et al.Deregulation of the NLRP3 inflammasome in hepatic parenchymal cells during liver cancer progression[J].Lab Invest,2014,94(1):52-62.
[17]Sonohara F,Inokawa Y,Kanda M,et al.Association of inflammasome components in background liver with poor prognosis after curatively-resected hepatocellular carcinoma[J].Anticancer Res,2017,37(1):293-300.
[18]Tanaka Y,Furuta T,Suzuki S,et al.Impact of interleukin-1b genetic polymorphisms on the development of hepatitis C virus-related hepatocellular carcinoma in Japan[J].J Inffect Dis,2003(187):1822-1825.
[19]Wang Y,Kato N,Hoshida Y,et al.Interleukin-1b gene polymorphisms associated with hepatocellular carcinoma in hepatitis C virus infection[J].Hepatology,2003,37(1):65-71.
[20]Hirankarn N,Kimkong I,Kummee P,et al.Interleukin-1beta gene polymorphism associated with hepatocellular carcinoma in hepatitis B virus infection[J].World J Gastroenterol,2006,12(5):776-779.
[21]Okamoto K,Ishida C,Ikebuchi Y,et al.The genotypes of IL-1 beta and MMP-3 are associated with the prognosis of HCV-related hepatocellular carcinoma[J].Intern Med,2010,49(10):887-895.
[22]Wei Q,Guo P,Mu K,et al.Estrogen suppresses hepatocellular carcinoma cells through ERbeta-mediated upregulation of the NLRP3 inflammasome[J].Lab Invest,2015,95(7):804-816.
[23]Fan SH,Wang YY,Lu J,et al.Luteoloside suppresses proliferation and metastasis of hepatocellular carcinoma cells by inhibition of NLRP3 inflammasome[J].PLoS One,2014,9(2):e89961.
[24]Shi X,Liu J,Ren L,et al.Nutlin-3 downregulates p53 phosphorylation on serine392 and induces apoptosis in hepatocellular carcinoma cells[J].BMB Rep,2014,47(4):221-226.
[25]Shi XL,Liu JL,Yu K,et al.Nutlin-3 promotes pyroptosis in SMMC-7721 cells[J].Chin J Immuno,2017,33(7):1014-1017.
[26]Yan JG.Radiofrequency ablation in the treatment of hepatocellular carcinoma:The observation of NLRP3 and IL-1β changes[J].J Clin Med,2017,4(28):5378.
[27]Jiang Z,Yao L,Ma H,et al.miRNA-214 inhibits cellular proliferation and migration in glioma cells targeting caspase 1 involved in pyroptosis[J].Oncol Res,2017,25(6):1009-1019.
[28]Zhang LL,Guo YJ,Zhao CN,et al.Effects and mechanism of miR-214 on hepatocellular carcinoma[J].Asian Pac J Trop Med,2015,8(5):392-398.
[29]Li H,Wang H,Ren Z.MicroRNA-214-5p inhibits the invasion and migration of hepatocellular carcinoma cells by targeting Wiskott-Aldrich syndrome like[J].Cell Physiol Biochem,2018,46(2):757-764.
[30]Wan J,Huang M,Zhao H,et al.A novel tetranucleotide repeat polymorphism within KCNQ1OT1 confers risk for hepatocellular carcinoma[J].DNA Cell Biol,2013,32(11):628-634.
[31]Li C,Miao R,Zhang J,et al.Long non-coding RNA KCNQ1OT1 mediates the growth of hepatocellular carcinoma by functioning as a competing endogenous RNA of miR-504[J].Int J Oncol,2018(52):1603-1612.
[32]Hu H,Yang L,Li L,et al.Long non-coding RNA KCNQ1OT1 modulates oxaliplatin resistance in hepatocellular carcinoma through miR-7-5p/ABCC1 axis[J].Biochem Biophys Res Commun,2018,503(4):2400-2406.
[33]Jin X,Jin H,Shi Y,et al.Long non-coding RNA KCNQ1OT1 promotes cataractogenesis via miR-214 and activation of the caspase-1 pathway[J].Cell Physiol Biochem,2017,42(1):295-305.
[34]Yang F,Qin Y,Wang Y,et al.LncRNA KCNQ1OT1 mediates pyroptosis in diabetic cardiomyopathy[J].Cell Physiol Biochem,2018,50(4):1230-1244.