［1］Ranganathan S，Krishnan A，Sivasithambaram ND.Significance of twist and iNOS expression in human breast carcinoma［J］.Mol Cell Biochem，2016，412(1-2)：41-47.
［2］Pazhang Y，Ahmadian S，Javadifar N，et al.COX-2 and survivin reduction may play a role in berberine-induced apoptosis in human ductal breast epithelial tumor cell line［J］.Tumour Biol，2012，33(1)：207-214.
［3］Fan GW，Gao XM，Wang H.The anti-inflammatory activities of Tanshinone IIa，an active component of TCM，are mediated by estrogen receptor activation and inhibition of iNOS［J］.J Steroid Biochem Mol Biol，2009，113(3-5)：275-280.
［4］Wink DA，Ridnour LA，Cheng R.The oncogenic properties of the redox inflammatory protein inducible nitric oxide synthase in ER(-) breast cancer［J］.Redox Biol，2015，5：413.
［5］Granados-Principal S，Liu Y，Guevara ML，et al.Inhibition of iNOS as a novel effective targeted therapy against triple-negative breast cancer［J］.Breast Cancer Res，2015，17：25.
［6］Lin CW，Shen SC，Ko CH，et al.Reciprocal activation of macrophages and breast carcinoma cells by nitric oxide and colony-stimulating factor-1［J］.Carcinogenesis，2010，31(12)：2039-2048.
［7］Abdin AA，Soliman NA，Saied EM.Effect of propranolol on IL-10，visfatin，Hsp70，iNOS，TLR2，and survivin in amelioration of tumor progression and survival in solid ehrlich carcinoma-bearing mice［J］.Pharmacol Rep，2014，66(6)：1114-1121.
［8］Zeybek ND，Inan S，Ekerbicer N.The effects of gemcitabine and vinorelbine on inducible nitric oxide synthase (iNOS) and endothelial nitric oxide synthase (eNOS) distribution of MCF-7 breast cancer cells［J］.Acta Histochem，2011，113(1)：62-67.
［9］Ban HS，Uno M，Nakamura H.Suppression of hypoxia-induced HIF-1alpha accumulation by VEGFR inhibitors：Different profiles of AAL993 versus SU5416 and KRN633［J］.Cancer Lett，2010，296(1)：17-26.
［10］Ichinoe M，Mikami T，Shiraishi H，et al.High micro vascular density is correlated with high VEGF iNOS and COX-2 expression in penetrating growth-type early gastric carcinomas［J］.Histopathology，2004，45：612-618.
［11］Sappayatosok K，Maneerat Y，Swasdison S，et al.Expression of pro-inflammatory protein，iNOS，VEGF and COX-2 in oral squamous cell carcinoma (OSCC)，relationship with angiogenesis and their clinico-pathological correlation［J］.Med Oral Patol Oral Cir Bucal，2009，14(7)：E319-324.
［12］Dabbeche-Bouricha E，Hadiji-Abbes N，Abdelmaksoud-Damak R，et al.Quantitative measurement of iNOS expression in melanoma，nasopharyngeal，colorectal，and breast tumors of Tunisian patients：comparative study and clinical significance［J］.Tumour Biol，2016，37(4)：5153-5164.
［13］Tschugguel W，Knogler W，Czerwenka K，et al.Presence of endothelial calcium-dependent nitric oxide synthase in breast apocrine metaplasia［J］.Br J Cancer，1996，74(9)：14-23.
［14］Sharon A Glynn，Brenda J Boersma，Tiffany H Dorsey，et al.Increased NOS2 predicts poor survival in estrogen receptor-negative breast cancer patients［J］.J Clin Invest，2010，120(11)：3843-3854.
［15］Meng Wen，Hou Gang，Jia Yusheng，et al.Significance and correlatioingship between inducible nitric oxide synthase and clinical pathologic parameter［J］.Chin J Cancer Prev Treat，2004，11(8)：808-810.［孟文，侯刚，贾玉生，等.诱导型一氧化氮合酶与乳腺癌临床病理的关系及意义［J］.肿瘤防治杂志，2004，11(8)：808-810.］
［16］Jin Z，Wang W，Jiang N，et al.Clinical implications of iNOS levels in triple-negative breast cancer responding to neoadjuvant chemotherapy［J］.PLoS One，2015，10(7)：e0130286.