[1]Tanaka TI,Alawi F.Human papillomavirus and oropharyngeal cancer[J].Dent Clin North Am,2018,62(1):111-120.
[2]Gannon OM,Antonsson A,Bennett IC,et al.Viral infections and breast cancer-A current perspective[J].Cancer Letters,2018(420):182-189.
[3]Elamrani A,Gheit T,Benhessou M,et al.Prevalence of mucosal and cutaneous human papillomavirus in Moroccan breast cancer[J].Papillomavirus Research,2018(5):150-155.
[4]Amador-Molina A,Trejo-Moreno C,Romero-Rodríguez D,et al.Vaccination with human papillomavirus-18 E1 protein plus α-galactosyl-ceramide induces CD8 cytotoxic response and impairs the growth of E1-expressing tumors[J].Vaccine,2019,37(9):1219-1228.
[5]Kantang W,Chunstrivirot S,Muangsin N,et al.Design of peptides as inhibitors of human papillomavirus 16 transcriptional regulator E1-E2[J].Chemical Biology & Drug Design,2016,88(4):475-484.
[6]Miaomiao Ma,Yaning Feng,Peiwen Fan,et al.Human papilloma virus E1-specific T cell immune response is associated with the prognosis of cervical cancer patients with squamous cell carcinoma[J].Infectious Agents and Cancer,2018,13(undefined):35.
[7]Michaelle Chojnacki,Thomas Melendy.The HPV E2 transcriptional transactivation protein stimulates cellular DNA polymerase epsilon[J].Viruses,2018,10(6):321.
[8]Zummeren MV,Kremer WW,Leeman A,et al.HPV E4 expression and DNA hypermethylation of CADM1,MAL,and miR124-2 genes in cervical cancer and precursor lesions[J].Modern Pathology,2018,31(12):1842-1850.
[9]Stevenson A,Kavanagh K,Pan J,et al.Risk stratification of cervical disease using detection of human papillomavirus (HPV) E4 protein and cellular MCM protein in clinical liquid based cytology samples[J].Journal of Clinical Virology,2018,108(undefined):19-25.
[10]Aldo Venuti,Francesca Paolini,Lubna Nasir,et al.Papillomavirus E5:the smallest oncoprotein with many functions[J].Molecular Cancer,2011,10(undefined):140.
[11]de Freitas AC,de Oliveira THA,Barros MR Jr,et al.hrHPV E5 oncoprotein:immune evasion and related immunotherapies[J].Journal of Experimental & Clinical Cancer Research:CR,2017,36(1):71.
[12]Christopher WW,Ethan LM.Human papillomavirus type 18 E5 oncogene supports cell cycle progression and impairs epithelial differentiation by modulating growth factor receptor signalling during the virus life cycle[J].Oncotarget,2017,8(61):103581-103600.
[13]F Belleudi,L Leone,V Purpura,et al.HPV16 E5 affects the KGFR/FGFR2b-mediated epithelial growth through alteration of the receptor expression,signaling and endocytic traffic[J].Oncogene,2011,30(50):4963-4976.
[14]Jung-Min Oh,Soo Sam Steve Kim,Eun-Ah Cho,et al.Human papillomavirus type 16 E5 protein inhibits hydrogen-peroxide-induced apoptosis by stimulating ubiquitin-proteasome-mediated degradation of Bax in human cervical cancer cells[J].Carcinogenesis,2010,31(3):402-410.
[15] Scott ML,Coleman DT,Kelly KC,et al.Human papillomavirus type 16 E5-mediated upregulation of Met in human keratinocytes[J].Virology,2018,519(undefined):1-11.
[16]G Hossein A,Mohammad H,Barbara M,et al.E5 protein of human papillomavirus 16 downregulates HLA class I and interacts with the heavy chain via its first hydrophobic domain[J].International Journal of Cancer,2010,119(9):2105-2112.
[17]Campo MS,Graham SV,Cortese MS,et al.HPV-16 E5 down-regulates expression of surface HLA class I and reduces recognition by CD8 T cells[J].Virology,2010,407(1):137-142.
[18] Estêvo D,Costa NR,Gil da Costa RM,et al.Hallmarks of HPV carcinogenesis:The role of E6,E7 and E5 oncoproteins in cellular malignancy[J].Biochimica et Biophysica Acta-Gene Regulatory Mechanisms,2019,1862(2):153-162.
[19]Mcbride AA,Warburton A.The role of integration in oncogenic progression of HPV-associated cancers[J].Plos Pathogens,2017,13(4):e1006211.
[20]Network TCGA.Integrated genomic and molecular characterization of cervical cancer[J].Nature,2017,543(7645):378-384.
[21]Martinezzapien D,Ruiz FX,Poirson J,et al.Structure of the E6/E6AP/p53 complex required for HPV-mediated degradation of p53[J].Nature,2016,529(7587):541-545.
[22]Maria F,Johnson MM,Marnelli B,et al.The large and small isoforms of human papillomavirus type 16 E6 bind to and differentially affect procaspase 8 stability and activity[J].Journal of Virology,2007,81(8):4116-4129.
[23]Xie X,Piao L,Bullock BN,et al.Targeting HPV16 E6-p300 interaction reactivates p53 and inhibits the tumorigenicity of HPV-positive head and neck squamous cell carcinoma[J].Oncogene,2014,33(8):1037-1046.
[24]Chiantore MV,Mangino G,Iuliano M,et al.Human papillomavirus E6 and E7 oncoproteins affect the expression of cancer-related microRNAs:additional evidence in HPV-induced tumorigenesis[J].Journal of Cancer Research & Clinical Oncology,2016,142(8):1751-1763.
[25]Yue Z,Ying H,Tian T,et al.MiR-21-5p,miR-34a,and human telomerase RNA component as surrogate markers for cervical cancer progression[J].Pathology-Research and Practice,2018,214(3):S0344033817310130.
[26]Shah M,Anwar MA,Park S,et al.In silico mechanistic analysis of IRF3 inactivation and high-risk HPV E6 species-dependent drug response[J].Scientific Reports,2015,5(1):13446.
[27] Pańczyszyn A,Boniewska-Bernacka E.Telomeres and telomerase during human papillomavirus-induced carcinogenesis[J].Molecular Diagnosis & Therapy,2018,22(4):1-10.
[28]Roman A,Munger K.The papillomavirus E7 proteins[J].Virology,2013,445(1-2):138-168.
[29]Fischer M,Uxa S,Stanko C,et al.Human papilloma virus E7 oncoprotein abrogates the p53-p21-DREAM pathway[J].Scientific Reports,2017,7(1):2603.
[30]Songock WK,Kim SM,Bodily JM.The human papillomavirus E7 oncoprotein as a regulator of transcription[J].Virus Research,2017(231):56-75.
[31]Hu H,Zhao J,Yu W,et al.Human papillomavirus DNA,HPV L1 capsid protein and p16 INK4a protein as markers to predict cervical lesion progression[J].Archives of Gynecology and Obstetrics,2019,299(1):141-149.
[32]Zheng L,Yanru WU,Wang Y.Clinical significance of HPV DNA and HPV L1 protein detection in cervical exfoliated cells[J].China Medical Herald,2016,13(5):107-110.
[33]Aksoy P,Gottschalk EY,Meneses PI.HPV entry into cells[J].Mutation Research/Reviews in Mutation Research,2016(772):13-22.
[34] Breiner B,Preuss L,Roos N,et al.Refolding and in vitro characterization of human papillomavirus 16 minor capsid protein L2[J].Biological Chemistry,2019,400(4):513-522.
[35]Campos SK.Subcellular trafficking of the papillomavirus genome during initial infection:The remarkable abilities of minor capsid protein L2[J].Viruses,2017,9(12):370.
[36]Mendoza L,Picconi MA,Mirazo S,et al.Distribution of HPV-16 variants among isolates from Paraguayan women with different grades of cervical lesion[J].International Journal of Gynaecology & Obstetrics the Official Organ of the International Federation of Gynaecology & Obstetrics,2013,122(1):44-47.
[37]Mosmann JP,Monetti MS,Frutos MC,et al.Mutation detection of E6 and LCR genes from HPV 16 associated with carcinogenesis[J].Asian Pac J Cancer Prev,2015,16(3):1151-1157.
[38]Emiko S,Ferran MC,Baker CC,et al.Repression of HPV16 early region transcription by the E2 protein[J].Virology,2006,351(1):29-41.
[39]Shailja P,Neeraj J,Prusty BK,et al.Human papillomavirus type 16 variant analysis of E6,E7,and L1 genes and long control region in biopsy samples from cervical cancer patients in north India[J].Journal of Clinical Microbiology,2008,46(3):1060.